Explaining why a specific outcome occurred, and which inputs deserve the blame or credit, is central to philosophical, scientific, and policy analysis. Existing tools split into two camps. The theory of actual causality (AC) gives principled verdicts, but only for toy-sized models, because computing them requires enumerating counterfactual scenarios. Scalable attribution methods like SHAP (or even causal SHAP) at least partially ignore the causal structure that generated the data, and can give answers that conflict with a careful causal analysis. We close this gap with Probabilistic Causal Impact (PCI). PCI builds on actual causality and on Pearl's notions of probability of necessity and sufficiency, but recasts the question of explainability as an estimation problem on a probabilistic causal model that is easily approximated via Monte Carlo. By specifying a distribution over "candidate explanations," a distribution over counterfactual values, and a scoring function, PCI provides tractable, causally grounded, graded explanations, generalizing AC and Pearl's probability of causation as degenerate cases. We evaluate PCI in synthetic and real-world examples, spanning consistency checks with AC, scaling experiments, complex continuous-valued dynamical systems, and a real-world deployed causal machine learning model trained on millions of datapoints.
The topic of explanation in recommender systems has seen steady research attention since the earliest days of the field. With some exceptions, this work has focused on the explanation of single items in a recommendation list and, especially recently, has emphasized approaches that are decoupled from the logic of the recommendation algorithm itself. Based on findings in the psychology of interpersonal communication, we propose a new task, pairwise interpretation of item rankings, asking the comparative question ``Why is item A ranked higher than item B?''. An effective solution to this task, we argue, is inherently grounded in the operation of the recommendation algorithm. We propose a class of techniques based on counterfactual learning to uncover the items in a user's profile that have contributed to the relative ranking of items. Using multiple datasets, we show that it is possible to identify such items as potential basis for comparative explanation.
This paper investigates the development of causal foundation models for bounding the effect of interventions and counterfactuals from observational data. We show that a canonical prior can be defined with full support over the space of structural causal models with discrete observables. With this canonical prior, we translate the problem of bounding counterfactuals into that of learning distributions over functions that map data (and possibly structural assumptions) to a causal query of interest. This extends the promising causal foundational modelling paradigm to the estimation of partially-identifiable causal effects, i.e., under unobserved confounding, where multiple values are equally compatible with the observed data and prior structural assumptions.
Generative models promise a route to explainable clinical AI: rather than probe a classifier, model the distributions of healthy and diseased patients and read explanations off the geometry between them. We build such a system - an optimal-transport rectified flow trained between two clinical distributions - and use it to ask a pointed question the field too rarely tests: do the resulting explanation heatmaps actually localize disease? On tabular tumour biomarkers (Breast Cancer Wisconsin) a single flow yields per-patient counterfactuals, an unsupervised malignancy score (AUROC 0.91; 0.93 +/- 0.01 across five seeds), and a label-free attribution that agrees with a supervised classifier (r ~ 0.5) - a compact, honest interpretability engine, though it never out-predicts logistic regression. Moving to chest X-rays, we show the transport heatmap is a population-level signal, not a localiser; a reconstruction-based, identity-preserving variant does localize synthetic lesions (pointing game 0.52), yet on real RSNA radiologist boxes it collapses to chance while only supervised Grad-CAM stays above it. The central result is a synthetic-to-real gap: label-free heatmaps that look compelling on planted lesions are not evidence of real localisation. We contribute a reusable optimal-transport recipe for generative explanations and a controlled benchmark for stress-testing whether they localize.
Gerrit Großmann, David A. Selby, Sebastian J. Vollmercs.AI
Structural causal models are the standard language for reasoning about interventions and counterfactuals, but they describe static variables, typically measured once, and usually forbid cyclic dependencies. Many systems we care about, such as patients, climates, and economies, instead evolve continuously in time, are observed at irregular time points, and contain feedback loops. We argue that neural operator learning provides a natural foundation for causal reasoning in this setting, and propose Orca, a framework in which each node of the causal graph is a function of time and each mechanism is a learned map between function spaces. We extend existing neural operator architectures to express causal mechanisms: a mechanism computes the function value of a node from its parent nodes by taking several parent functions as input, respects the arrow of time, and treats latent exogenous noise as a function that can be inferred and reused for counterfactuals. We formalize the model class and demonstrate counterfactual reasoning on synthetic continuous-time examples. Code is available at https://github.com/gerritgr/orca
Vahidin Hasic, Chao Wang, Luis C. Garcia-Peraza-Herrera +2cs.CV cs.AI
Multimodal large language models (MLLMs) increasingly support high-stakes decision making by combining complementary information from images and text. While existing explainability methods identify influential image regions or text tokens, they cannot answer a fundamental question: which modality drives a prediction? Consequently, a model may produce the correct output while relying on the wrong source of evidence, masking shortcut learning and unsafe reasoning. We formulate modality attribution as a complementary explainability objective for multimodal foundation models and propose Counterfactual Modality Attribution (CMA), the first framework for quantifying modality-level contributions in MLLMs. CMA generates image-only, text-only, and joint multimodal counterfactuals using coupled diffusion priors and converts them into principled modality attribution scores through a cooperative game-theoretic formulation based on Shapley values. We evaluate CMA on controlled synthetic benchmarks with known ground-truth modality reliance and on a real-world multimodal clinical dataset. CMA correctly identifies the decision-driving modality in 98% of controlled cases and consistently outperforms baselines, revealing failures of cross-modal reasoning that remain invisible to predictive accuracy alone. Our results establish modality attribution as a complementary dimension of explainability beyond feature attribution, providing a principled framework for auditing multimodal foundation models in safety-critical applications.
Dennis Thumm, Billy Tim Anthony, Ying Chencs.LG physics.data-an stat.ME
Most benchmarks for causal inference over time series are observational, small, or domain-specific, leaving interventional and counterfactual estimation under-served exactly where it matters most, such as in healthcare, policy evaluation, and climate science. We introduce \textbf{DoTime}, an open, scalable, and theoretically grounded generator of multivariate temporal structural causal models (TSCMs) with interventions, released as the \code{dotime} PyPI package together with four frozen evaluation suites. Beyond existing work, it adds capabilities absent from prior generators: continuous-time intervention \emph{windows}, counterfactual sampling modes with a positivity guard, regime-switching SCMs as a strict generalization of interrupted time series, non-stationary dynamics by construction with switching SCM parameters, and deterministic ramp and sinusoidal intervention profiles that place trends and structural breaks \emph{inside} the evaluation window. Moreover, it demonstrates the suitability of the generator as a prior for a causal foundation model reference implementation. The released suites span a training-scale snapshot of $100{,}000$ trajectories and eight named identification structures, each with exact ground truth: paired interventional trajectories from the same SCM throughout, and shared-noise counterfactuals in the continuous-time suite. We ship reference baseline implementations with an evaluation harness, and pose a falsifiable claim: interventional training buys a measurable direction-accuracy advantage over an observational model of identical capacity. It is tested across three training seeds per arm. Under structure-matched evaluation on held-out episodes, the interventional prior-fitted network's (PFN) gap is positive in every structure, trajectory length, and seed tested.
Decision theory provides a formal framework for how agents should make choices under uncertainty, drawing on ideas from philosophy, probability, and causality. Despite significant progress, the field still lacks a unified modeling language, and key concepts - such as the distinction between subjective and objective elements, or what it means for a decision theory to perform well - are often left implicit. This can make it difficult to evaluate and compare competing theories, particularly in controversial cases. In this paper, we address these issues by introducing a formal framework for decision theory based on nonparametric structural equation models (NPSEMs), a well-established tool in causal inference. NPSEMs provide a unified foundation for representing agents, counterfactuals, and causal relationships, allowing for unambiguous definitions of EDT and CDT. Building on this foundation, we propose a novel decision theory - personal decision theory - which instructs agents to maximize a subjective model of their own counterfactual utility. We introduce a formal performance metric based on hypothetical interventions that enforce a given decision theory across a population - such as might be achieved through education or policy -- and show that, under certain assumptions, personal decision theory is optimal with respect to this metric. Throughout, we use the smoking lesion problem as a running example and conclude with a formal analysis of Newcomb's problem. Our aim is to provide decision theory with a clearer modeling language and firmer evaluative ground, thereby enabling more rigorous comparisons and facilitating conceptual progress in the field.
Over a series of seven papers, Andreas & Günther have introduced seven definitions of actual causation and have classified them as belonging to three different, competing, types of accounts: factual difference-making, counterfactual difference-making, and regularity-based. I show that their most recent - factual difference-making - definition instantiates all three types, thereby proving that these are distinctions without a difference. I further compare their novel account to the other six accounts on several crucial examples, revealing that this undermines all seven of their accounts.
A common assumption holds that enough observational and interventional data, given to a strong enough predictor, suffices. We report a failure mode that contradicts it. Across hundreds of structural causal models, on identified quantities a strong predictor and a Bayesian baseline both succeed, but on unidentified quantities (the couplings between counterfactual worlds) the predictor collapses to a point, on 28% of models to one no valid model can produce, while the truth is an admissible interval more data never narrows. The gap is structural: prediction cannot represent uncertainty over counterfactual couplings. We cast a world model as a single positive semidefinite coupling kernel K(T,T') over admissible worlds, whose diagonal is the ordinary posterior (what a predictor recovers) and whose off-diagonal is the cross-world coupling it cannot, which every counterfactual reads. The paper is the theory of that off-diagonal. It is real: two states with identical posteriors differ on a cross-world query, and the off-diagonal is the coupling that fixes counterfactuals. It can be bounded: positive semidefiniteness is partial-identifying information the marginals lack, and enforcing it bounds counterfactuals in polynomial time where the exact response-type program is intractable. Logical structure sharpens it: ontology axioms tighten the bound by up to a third, propagating to couplings they never touch. It can be acquired: targeted scars, constraints learned from encountered infeasibilities, close the gap several times faster than untargeted ones. Its full reconstruction is approximate counting of the admissible worlds, tractable below the Sly-Sun threshold and inapproximable above; we do not claim to beat the worst case.
Stories hold a reader's attention because they have causes, secrets, and consequences. Shadow-Loom is an experimental open-source framework that turns a narrative into a versioned graphical world model and lets two engines act on it: a causal physics grounded in Pearl's ladder of causation and a recently proposed counterfactual calculus over Ancestral Multi-World Networks; and a narrative physics that scores the same graph against four structural reader-states -- mystery, dramatic irony, suspense, and surprise -- in the tradition of Sternberg's curiosity/suspense/surprise triad, with suspense formalised in the structural-affect line of work on story comprehension and computational suspense. Large language models are used only at the boundary: extraction, rendering, and audit; identification, intervention, and counterfactual reasoning are carried out in typed code over the graph. The system is offered as a research artefact rather than as a benchmarked NLP model; code, fixtures, and pipeline are released open source.