We give a negative solution to MAIS-O60. We first construct an example in which an initially active ReLU neuron becomes completely inactive in finite time and thereafter remains frozen at a limit whose Fourier energy is equally distributed among all nonzero real frequency classes. The counterexample holds on an open set of initial conditions and therefore occurs with positive probability under Gaussian initialization. An appendix prepared by GPT-5.6 Sol strengthens the counterexample by showing that the same failure can occur for every Clarke trajectory from an open set of initial conditions, under the convention $\mathrm{ReLU}'(0)=0$, for smooth dead-zone approximations of ReLU, and for fixed-step full-batch gradient descent. Thus, single-frequency alignment is not a general consequence of training a single neuron on modular addition.
Grokking -- generalization long after training-set interpolation -- has been accelerated by structure-agnostic interventions (gradient filtering, weight-norm clamping, geometric penalties). Whether the delay specifically measures the time to form task-structured representations has remained observational. We test it causally by injecting representational priors of varying content into a one-layer transformer learning modular addition, via a supervised-contrastive loss whose positives encode (i) the task's true structure ($(a+b) \bmod p$), (ii) a coherent-but-wrong sibling ($(a-b) \bmod p$), or (iii) a random partition -- all with identical loss form, strength, class sizes, and geometry. Whether generalization occurs follows a clean gradation: true 22/30 runs, sibling (same periodic features, wrong combination) 14/15, random (only memorizable) 0/20 (Fisher $p=1.3\times10^{-7}$). A weight-norm-matched control replaying the norm trajectory onto plain cross-entropy generalizes 0/15, ruling out the norm as mediator. Probes show structure formation precedes and predicts generalization in all runs. Only the true structure also accelerates grokking (up to $2.75\times$), but this is dose-dependent and bimodal. We then confirm the mechanism by prediction: because the acceleration is gated by a weight-norm side-effect, clamping the norm during training yields a reliable, standalone accelerator with a median $8.6\times$ speedup (up to $22\times$ on the fastest seeds, under 1000 epochs), growing monotonically as the norm is held lower; the residual stalls also vanish, though significant only pooled over the two mitigations run at both strengths ($0/40$ vs $6/20$, $p=7.7\times10^{-4}$), not per method. The grokking delay is, causally, the time to form the right representational structure -- decided at the level of features, not labels.